hackney

jak juz chcecie ta kozieradke to kupcie cos z ''gornej połki'', polecam test freak, nic mocniejszego nie znajdziesz;)

Ty masz to po bupropionie a nie kozieradce, ogarnij sie;)

Libido – It Really Is All in Your Head Treating low libido with hormones often falls short because it neglects one of the most important organs for a healthy sex life … your brain. The components of healthy libido — desire, arousal, orgasm, and resolution — are all controlled by brain chemicals called neurotransmitters. In order for you to improve your sex drive, you need to optimize the following neurotransmitters: Dopamine for Desire Acetylcholine for Arousal GABA for Orgasms Serotonin for Resolution Let’s take a look at some natural strategies for boosting these important brain chemicals and improving libido. Restore Sex Drive with Dopamine It’s now well-established that dopamine affects sexual desire. As a matter of fact, medications that block dopamine receptors have been associated with decreased libido. The good news is that libido can improve with the right combination of vitamins, minerals, natural treatments. Here are a few suggestions for boosting brain dopamine levels:3 Amino acids tyrosine and phenylalanine Folic acid and thiamine (B1) Chromium Rhodiola rosea Ginkgo biloba Boost Sexual Arousal with Acetylcholine According to Dr. Braverman, the brain chemical acetylcholine is responsible for arousal. It’s believed that people with low brain levels of acetylcholine are unable to concentrate on sex, hence disrupting the normal arousal process.4 For this reason, to improve arousal, you might consider the following acetylcholine boosters:3 Choline DMAE Acetyl-L-carnitine Phosphatidylserine Omega-3 fatty acids Manganese Enhance Orgasms with GABA GABA is responsible for regulating sexual tone and plays an important role in overall brain function. Therapeutic drugs that increase the amount of GABA in the brain typically have relaxing, anti-anxiety, and mood-soothing effects.5,6 Conversely, when GABA becomes depleted, it is difficult to relax and let go of fear and negative feelings. Hence, orgasm becomes difficult. GABA-enhancing compounds include the following:3 GABA (available as a supplement) Inositol B Vitamins Kava kava Branched-chain amino acids (leucine, isoleucine, and valine) Improve Sexual Resolution with Serotonin The final phase of libido is resolution. This is established and maintained by the brain chemical serotonin, which modulates anger, aggression, body temperature, mood, sleep, and sexuality.7 Low serotonin levels can result in a lack of joy and decreased feelings of intimacy. Without resolution, intimacy is lost. This can disrupt future sexual desire and arousal. If sexual intimacy and joy are issues for you, try optimizing brain serotonin levels.3 Here are some nutrients that can help: Tryptophan 5-HTP Saffron extract Omega-3 fatty acids Magnesium

ponawiam: Sylvek a jakie dawki cebergoliny/ropinirolu i buspironu stosowac? i nie pisac o dostepie bo mam chyba wszystko co mozliwe na pssd

Sylvek a jakie dawki cebergoliny/ropinirolu i buspironu stosowac?

Od kilku dni biore buspiron 30mg. Powrocily spontaniczne erekcje wilgotność członka nie mam żadnych problemów z erekcją. No i jak stanie to jest na prawdę twardy;-) -- 03 kwi 2013, 14:31 -- po buspironie bardzo mnie mulilo i musialem odstawic, teraz zaczalem brac amantadyne no i zobaczymy co z tego wyjdzie..

Barsinister a jak po mirtazapinie?

od kilku dni widzę dużą poprawę w libido, przy masturbacji zajmuje mi to doslownie moment, a jeszcze niedawno nie moglem dojsc, a erekcje tez bylo trudno. Zaczynam się podniecac i napalac, no i w koncu zaczynam miec z tego przyjemnosc:) Spermy wiecej, gestsza i zapach jak kiedys;) Biore obecnie testosteron i moklobemid. Nie wiem które pomoglo, test biore od 6ciu tyg a moklo od 2. Mam nadzieje ze to od moklo, bo gdyby od tescia to juz wczesniej powinno sie to pojawic hmm. Tak dobrze juz dawno nie było:):):)

sylvek napisz cos wiecej o leczeniu PSSD, jakie srodki i dawki. To ważne bo widze ze jestes ogarniety w temacie i mozesz pomoc nam

sylvek napisz cos wiecej o leczeniu PSSD, jakie srodki i dawki. To ważne bo widze ze jestes ogarniety w temacie i mozesz pomoc nam

te leki mogą powodowac spadek oksytocyny odpowiedzialnej za przywiązanie do partnera, podniecenie i właśnie nawilzenie pochwy. Jeden z uzytkowników piszacy posty wyzej próbował oksytocyny i mówil ze czuł sie bardziej przywiazany do partnerki, czul wieksza bliskosc.

te leki mogą powodowac spadek oksytocyny odpowiedzialnej za przywiązanie do partnera, podniecenie i właśnie nawilzenie pochwy. Jeden z uzytkowników piszacy posty wyzej próbował oksytocyny i mówil ze czuł sie bardziej przywiazany do partnerki, czul wieksza bliskosc.

bupropion próbowalem w dawkach 150mg nie dzialal, a powyzej powodowal odrealnienie. wzialem nebi zeby poprawic samopoczucie na buspironie Sexual side effects SSRIs, especially escitalopram, can cause various types of sexual dysfunction such as anorgasmia, erectile dysfunction, and diminished libido. Initial studies found that such side effects occur in more than 80% of patients, but since these studies relied on unprompted reporting, the frequency was probably overestimated. In more recent studies, doctors have specifically asked about sexual difficulties, and found that they are present in between 17% and 41% of patients. Stimulation of postsynaptic 5-HT2 and 5-HT3 receptors decreases dopamine and norepinephrine release from the substantia nigra. A number of drugs are not associated with sexual side effects (bupropion, mirtazapine (Remeron), maprotiline (Ludiomil), (some of these are also not associated with weight gain). As a result, sexual dysfunction caused by SSRIs can sometimes be mitigated by several different drugs. These include: bupropion (norepinephrine and dopamine reuptake inhibitor) buspirone (serotonin 5-HT1A receptor partial agonist) cyproheptadine (5-HT2 antagonist) methylphenidate (stimulant) mirtazapine (noradrenergic and specific serotonergic antidepressant (NaSSA)) amphetamine (stimulant) amantadine (antiviral drug) pramipexole (dopamine agonist) and ropinirole (dopamine agonist) -- 23 lut 2013, 12:39 -- A variety of antidotes have been reported to treat SSRI-induced sexual dysfunction effectively; however, virtually all the data on these agents are derived from open case reports and case series. Insofar as sexual function improvement may be responsive to placebo effects, it is impossible to estimate the true efficacy of these antidotes.[27] Most of these antidotes either have serotonin-blocking properties (especially 5HT-2 antagonistic effects) or augment catecholamine activity, especially that of dopamine. The antiserotonergic antidotes are cyproheptadine, buspirone, nefazodone, and mianserin. Medications enhancing dopaminergic tone include amantadine, bupropion, and stimulants, with yohimbine showing noradrenergic effects. Among the reported antidotes, the only 2 without antiserotonergic effects or catecholaminergic activity are gingko biloba and urecholine. Cyproheptadine is an antihistamine with antiserotonergic properties that has been reported for over a decade to reverse antidepressant-induced sexual dysfunction. Only case reports and case series attest to its efficacy.[13,42-44] Effective doses range from 2mg to 16mg. In the most recent and largest case series, 12 of 25 patients described improvement in sexual function when treated with cyproheptadine (mean dose, 8.6mg).[13] Anorgasmia is the sexual side effect most often reported to be alleviated by cyproheptadine. Cyproheptadine is effective when taken either on an as-needed basis (typically, 1 to 2 hours before intercourse) or on a regular basis. However, cyproheptadine's utility is often limited by its potential side effects. Excessive sedation and the reversal of the therapeutic effect of the antidepressant are major problems that limit its usefulness. Effectively treated depression and bulimic symptoms have been reported to reemerge soon after cyproheptadine was started.[42,45-48] This reversal of therapeutic effects is itself reversible upon discontinuation. Buspirone is a serotonin-IA partial agonist typically prescribed to treat persistent anxiety. One case series reported that buspirone reversed both decreased sexual interest and orgasmic dysfunction caused by SSRIs.[49] Most patients using buspirone to treat sexual dysfunction take it daily. The dosage is the same as that used for anxiety (15mg to 60mg daily). The mechanism of action of buspirone in treating sexual dysfunction may be reduction of serotonergic tone via stimulation of presynaptic autoreceptors or the alpha-2 antagonist effects of one of buspirone's major metabolites, 1-pyrimidinylpiperazine. Nefazodone and mianserin are antidepressants with strong postsynaptic blocking properties. In one case report, nefazodone 150mg taken 1 hour prior to sexual activity completely reversed sertraline-induced anorgasmia.[50] Mianserin, an antidepressant with 5HT-2 and alpha-2 adrenergic antagonist properties, is available in many countries but not in the US. It has been reported to reverse serotonin reuptake inhibitor-induced sexual dysfunction in 9 of 15 patients.[51] Mirtazapine is similar in its biological activity to mianserin and might also be effective in reversing sexual side effects. No case reports or case series have yet been published attesting to this, although clinicians have described such an effect. The putative capacity of mianserin and mirtazapine to reverse sexual side effects can be attributed either to their serotonergic activity or presynaptic alpha-2 activity. Amantadine, a dopamine agonist, is used both as an antiviral agent and as a treatment for Parkinson's disease. It has been shown in a number of small case series to reverse anorgasmia.[13,52-54] Reported effective doses have ranged between 100mg to 400mg taken either on a daily or as-needed basis. In the most recent case series, 8 (42%) out of 19 patients with SSRI-induced sexual dysfunction improved with amantadine 200mg daily.[13] Given dopamine's consistent effect as a neurotransmitter involved in sexual arousal, a number of other dopamine agonists have been explored as treatments for sexual side effects.[2,55,56] Bupropion is another commonly touted antidote for SSRI-induced sexual dysfunction.[57,58] It is assumed that the mechanism of action by which bupropion reverses sexual side effects is its weak dopamine agonism. The evidence for bupropion's efficacy is scant, except for unpublished, anecdotal reports, one case report,[57] and a case series[58] in which 31 (66%) of 47 patients showed improvement when bupropion was added to the regimen along with the serotonergic antidepressant. Most patients (18/31) with a successful outcome responded to as-needed use of bupropion 75mg to 150mg. Libido, arousal, and orgasmic difficulties were all effectively reversed. Fifteen percent of treated patients stopped taking bupropion because of its stimulation side effects. It is unclear whether bupropion doses need to be somewhat lower than usual when added to fluoxetine or paroxetine, to compensate for pharmacokinetic interactions resulting in increased bupropion levels.[59] Stimulants, such as methylphenidate, D-amphetamine, and pemoline, are reported to reverse a variety of sexual side effects caused by SSRIs or MAOIs.[60-62] Low doses of 10mg-25mg of methylphenidate or D-amphetamine have been effective. One should add stimulants to an MAOI with extreme caution because of the risk of a hypertensive episode. However, use of an MAOI/stimulant combination has been shown to be safe in a case series.[63] SSRI/stimulant combinations show no similar risks. Yohimbine is available with or without a prescription (and with unclear purity) in health food stores. It is an alkaloid from the bark of Corynanthe yohimbi (family, Rubiaceae) and has been used for decades to reverse erectile dysfunction.[64-66] Its efficacy in treating sexual dysfunction may be associated with its ability to block presynaptic alpha-2 adrenergic sites, leading to enhanced adrenergic tone.[65] A variety of sexual side effects have been reported to be alleviated by yohimbine in doses ranging from 2.7mg to 16.2mg daily, prescribed either on a regular 5.4mg 3 times daily basis or on an as-needed basis with single doses up to 16.2mg.[13,67-69] In the largest case series, 17 (81%) of 21 patients showed improvement of sexual side effects when treated with yohimbine (mean dose, 16.2mg).[12] Typical side effects associated with yohimbine include anxiety, nausea, flushing, urinary urgency, and sweating. Yohimbine has been the subject of the only double-blind, placebo-controlled study to evaluate treatment of sexual dysfunction occurring as a drug side effect.[27] Unfortunately, the placebo effect was marked, showing a minimal drug-placebo difference with yohimbine given at a dose of 5.4mg 3 times daily. Yohimbine is also available in lower potency without a prescription. The purity, potency, and safety of these preparations, however, are unknown. Bethanechol is a cholinergic agonist that has occasionally been useful in reversing sexual dysfunction associated with TCAs and MAOIs.[70-73] Typical doses are 10mg to 20mg as needed or 30mg to 100mg daily in a divided dose. Potential side effects with bethanechol include diarrhea, cramps, and diaphoresis. No reports have evaluated or suggested the efficacy of bethanechol for treating SSRI-induced sexual side effects. Gingko biloba is an herbal extract reported to reverse a variety of sexual dysfunctions associated with antidepressants. Information about gingko's ability in this regard is derived from the experience of 1 clinician presenting a large case series.[74] The response rate was greater than 80%, with doses ranging from 60mg twice daily to 120mg twice daily (mean daily dose, 207mg). Reported side effects include gastrointestinal upset, lightheadedness, and stimulation effects. Because gingko may inhibit platelet-activating factor, caution should be used in considering its use by any patient with a bleeding diathesis. The mechanism by which gingko might alleviate sexual dysfunction is unknown.

bupropion próbowalem w dawkach 150mg nie dzialal, a powyzej powodowal odrealnienie. wzialem nebi zeby poprawic samopoczucie na buspironie Sexual side effects SSRIs, especially escitalopram, can cause various types of sexual dysfunction such as anorgasmia, erectile dysfunction, and diminished libido. Initial studies found that such side effects occur in more than 80% of patients, but since these studies relied on unprompted reporting, the frequency was probably overestimated. In more recent studies, doctors have specifically asked about sexual difficulties, and found that they are present in between 17% and 41% of patients. Stimulation of postsynaptic 5-HT2 and 5-HT3 receptors decreases dopamine and norepinephrine release from the substantia nigra. A number of drugs are not associated with sexual side effects (bupropion, mirtazapine (Remeron), maprotiline (Ludiomil), (some of these are also not associated with weight gain). As a result, sexual dysfunction caused by SSRIs can sometimes be mitigated by several different drugs. These include: bupropion (norepinephrine and dopamine reuptake inhibitor) buspirone (serotonin 5-HT1A receptor partial agonist) cyproheptadine (5-HT2 antagonist) methylphenidate (stimulant) mirtazapine (noradrenergic and specific serotonergic antidepressant (NaSSA)) amphetamine (stimulant) amantadine (antiviral drug) pramipexole (dopamine agonist) and ropinirole (dopamine agonist) -- 23 lut 2013, 12:39 -- A variety of antidotes have been reported to treat SSRI-induced sexual dysfunction effectively; however, virtually all the data on these agents are derived from open case reports and case series. Insofar as sexual function improvement may be responsive to placebo effects, it is impossible to estimate the true efficacy of these antidotes.[27] Most of these antidotes either have serotonin-blocking properties (especially 5HT-2 antagonistic effects) or augment catecholamine activity, especially that of dopamine. The antiserotonergic antidotes are cyproheptadine, buspirone, nefazodone, and mianserin. Medications enhancing dopaminergic tone include amantadine, bupropion, and stimulants, with yohimbine showing noradrenergic effects. Among the reported antidotes, the only 2 without antiserotonergic effects or catecholaminergic activity are gingko biloba and urecholine. Cyproheptadine is an antihistamine with antiserotonergic properties that has been reported for over a decade to reverse antidepressant-induced sexual dysfunction. Only case reports and case series attest to its efficacy.[13,42-44] Effective doses range from 2mg to 16mg. In the most recent and largest case series, 12 of 25 patients described improvement in sexual function when treated with cyproheptadine (mean dose, 8.6mg).[13] Anorgasmia is the sexual side effect most often reported to be alleviated by cyproheptadine. Cyproheptadine is effective when taken either on an as-needed basis (typically, 1 to 2 hours before intercourse) or on a regular basis. However, cyproheptadine's utility is often limited by its potential side effects. Excessive sedation and the reversal of the therapeutic effect of the antidepressant are major problems that limit its usefulness. Effectively treated depression and bulimic symptoms have been reported to reemerge soon after cyproheptadine was started.[42,45-48] This reversal of therapeutic effects is itself reversible upon discontinuation. Buspirone is a serotonin-IA partial agonist typically prescribed to treat persistent anxiety. One case series reported that buspirone reversed both decreased sexual interest and orgasmic dysfunction caused by SSRIs.[49] Most patients using buspirone to treat sexual dysfunction take it daily. The dosage is the same as that used for anxiety (15mg to 60mg daily). The mechanism of action of buspirone in treating sexual dysfunction may be reduction of serotonergic tone via stimulation of presynaptic autoreceptors or the alpha-2 antagonist effects of one of buspirone's major metabolites, 1-pyrimidinylpiperazine. Nefazodone and mianserin are antidepressants with strong postsynaptic blocking properties. In one case report, nefazodone 150mg taken 1 hour prior to sexual activity completely reversed sertraline-induced anorgasmia.[50] Mianserin, an antidepressant with 5HT-2 and alpha-2 adrenergic antagonist properties, is available in many countries but not in the US. It has been reported to reverse serotonin reuptake inhibitor-induced sexual dysfunction in 9 of 15 patients.[51] Mirtazapine is similar in its biological activity to mianserin and might also be effective in reversing sexual side effects. No case reports or case series have yet been published attesting to this, although clinicians have described such an effect. The putative capacity of mianserin and mirtazapine to reverse sexual side effects can be attributed either to their serotonergic activity or presynaptic alpha-2 activity. Amantadine, a dopamine agonist, is used both as an antiviral agent and as a treatment for Parkinson's disease. It has been shown in a number of small case series to reverse anorgasmia.[13,52-54] Reported effective doses have ranged between 100mg to 400mg taken either on a daily or as-needed basis. In the most recent case series, 8 (42%) out of 19 patients with SSRI-induced sexual dysfunction improved with amantadine 200mg daily.[13] Given dopamine's consistent effect as a neurotransmitter involved in sexual arousal, a number of other dopamine agonists have been explored as treatments for sexual side effects.[2,55,56] Bupropion is another commonly touted antidote for SSRI-induced sexual dysfunction.[57,58] It is assumed that the mechanism of action by which bupropion reverses sexual side effects is its weak dopamine agonism. The evidence for bupropion's efficacy is scant, except for unpublished, anecdotal reports, one case report,[57] and a case series[58] in which 31 (66%) of 47 patients showed improvement when bupropion was added to the regimen along with the serotonergic antidepressant. Most patients (18/31) with a successful outcome responded to as-needed use of bupropion 75mg to 150mg. Libido, arousal, and orgasmic difficulties were all effectively reversed. Fifteen percent of treated patients stopped taking bupropion because of its stimulation side effects. It is unclear whether bupropion doses need to be somewhat lower than usual when added to fluoxetine or paroxetine, to compensate for pharmacokinetic interactions resulting in increased bupropion levels.[59] Stimulants, such as methylphenidate, D-amphetamine, and pemoline, are reported to reverse a variety of sexual side effects caused by SSRIs or MAOIs.[60-62] Low doses of 10mg-25mg of methylphenidate or D-amphetamine have been effective. One should add stimulants to an MAOI with extreme caution because of the risk of a hypertensive episode. However, use of an MAOI/stimulant combination has been shown to be safe in a case series.[63] SSRI/stimulant combinations show no similar risks. Yohimbine is available with or without a prescription (and with unclear purity) in health food stores. It is an alkaloid from the bark of Corynanthe yohimbi (family, Rubiaceae) and has been used for decades to reverse erectile dysfunction.[64-66] Its efficacy in treating sexual dysfunction may be associated with its ability to block presynaptic alpha-2 adrenergic sites, leading to enhanced adrenergic tone.[65] A variety of sexual side effects have been reported to be alleviated by yohimbine in doses ranging from 2.7mg to 16.2mg daily, prescribed either on a regular 5.4mg 3 times daily basis or on an as-needed basis with single doses up to 16.2mg.[13,67-69] In the largest case series, 17 (81%) of 21 patients showed improvement of sexual side effects when treated with yohimbine (mean dose, 16.2mg).[12] Typical side effects associated with yohimbine include anxiety, nausea, flushing, urinary urgency, and sweating. Yohimbine has been the subject of the only double-blind, placebo-controlled study to evaluate treatment of sexual dysfunction occurring as a drug side effect.[27] Unfortunately, the placebo effect was marked, showing a minimal drug-placebo difference with yohimbine given at a dose of 5.4mg 3 times daily. Yohimbine is also available in lower potency without a prescription. The purity, potency, and safety of these preparations, however, are unknown. Bethanechol is a cholinergic agonist that has occasionally been useful in reversing sexual dysfunction associated with TCAs and MAOIs.[70-73] Typical doses are 10mg to 20mg as needed or 30mg to 100mg daily in a divided dose. Potential side effects with bethanechol include diarrhea, cramps, and diaphoresis. No reports have evaluated or suggested the efficacy of bethanechol for treating SSRI-induced sexual side effects. Gingko biloba is an herbal extract reported to reverse a variety of sexual dysfunctions associated with antidepressants. Information about gingko's ability in this regard is derived from the experience of 1 clinician presenting a large case series.[74] The response rate was greater than 80%, with doses ranging from 60mg twice daily to 120mg twice daily (mean daily dose, 207mg). Reported side effects include gastrointestinal upset, lightheadedness, and stimulation effects. Because gingko may inhibit platelet-activating factor, caution should be used in considering its use by any patient with a bleeding diathesis. The mechanism by which gingko might alleviate sexual dysfunction is unknown.

z reka i kobieta to samo jest;p jeszcze miesiac temu jak bralem buspiron to moglem szczytowac raz za razem, potem wzialem nebivolol i wszystko schrzanil. Zaczalem brac moklo jako antidotum ale nawet pogorszylo sie:( buspiron przestalem brac bo zle dzialal na psychike

z reka i kobieta to samo jest;p jeszcze miesiac temu jak bralem buspiron to moglem szczytowac raz za razem, potem wzialem nebivolol i wszystko schrzanil. Zaczalem brac moklo jako antidotum ale nawet pogorszylo sie:( buspiron przestalem brac bo zle dzialal na psychike

biore moklobemid, na pssd nie pomaga, moze troche bardziej sie chce, ale nie moge na nim dojsc,jak juz zbliza sie orgazm to palka opada i koniec, a biore dodatkowo testosteron;p. Pomaga na lęki i samopoczucie za to:)

biore moklobemid, na pssd nie pomaga, moze troche bardziej sie chce, ale nie moge na nim dojsc,jak juz zbliza sie orgazm to palka opada i koniec, a biore dodatkowo testosteron;p. Pomaga na lęki i samopoczucie za to:)

kamil piszesz o tym co brales a nie piszesz nic o odbloku. moze sprubój zrobic odblok i po nim sprawdz jaki bedzie poziom testo

Brałem też nebivolol 2 dni po 1/4 i 1 dzien 1/2 tabl i miekka faja od razu:/ Tylko 3 dni, w sumie jedna tabl i taka lipa:( biore tez buspiron, który juz zaczynal fajnie dzialac, ale pokusilem sie o dodanie tego nebivololu i teraz juz nie czuje tego buspironu echhh tymi lekami mozna tak samo szybko sobie pomóc jak zaszkodzic... Zacząłem injekcje testosteronem z tego wszystkiego, idzie propek na kicka i cypek w tle, do tego bedzie HCG zapobiegawczo -- 28 sty 2013, 21:28 -- Oto co dziś wyczytałem: ''Jednym z istotnym zastrzeżeń wysuwanych w stosunku do b-adrenolityków są zaburzenia funkcji seksualnych wywoływane lub nasilane przez te leki. Jedynym wyjątkiem w swojej klasie jest właśnie nebiwolol, który nie pogarsza, ale nawet może korzystnie modyfikować zaburzenia erekcji i dysfunkcję seksualną. Ważne jest również, że można go kojarzyć z sildenafilem. Jako jedyny z b-adrenolityków został wymieniony w zaleceniach Europejskiego Towarzystwa Nadciśnienia Tętniczego jako lek korzystnie działający w przypadku zaburzeń erekcji.'' To o co tu k...a chodzi?!?!?! Za małe dawki czy jaki h.j?!?!?! już mam dosyc po prostu...

bromokryptyna wg mnie słaba, chyba że przy dużych dawkach, ale sajdy wtedy chyba wystąpią, na mnie słabo działała, może też dlatego że zbyt nisko wtedy zbija prolaktyne? Queen jakie dawkowanie byś polecił? Piribedil działał ale sajdy masakryczne miałem, telepotałem się z zimna, pocilem się, drgawki i wielka sennośc!!! -- 01 lis 2012, 10:55 -- a z tymi beta-blokerami to nie jest na odwrót??? Jak wpisac w google np.''propranolol libido'' to wszędzie wyskakuje info o impotencji i zmniejszeniu libido!!! Wiem że coś z tą noradrenalina jest na rzeczy bo nawet jak biorę spalacza tluszczu co wlaśnie powoduje zwiekszenie norki to libido idzie mi w góre i erekcja też.

Hej jestem tu nowy i mam podobny problem jak Wy. Moje problemy z PSSD zaczęły się dosyc wcześnie bo już po pierwszych dawkach CITALU, potem już było tylko gorzej. Myślałem, że to nie ważne, że jak odstawię leki to to minie, a najważniejsze to wkońcu naprawic sobie psychikę, a nie dośc że te SSRI nie pomoghły za wiele w depresji to dodatkowo spowodowały silne PSSD nawet po odstawieniu. Brałem citalopram, escitalopram, fluoksetyne, wenlafaksyne. Potem natrafilem na Wellbutrin XR i dopiero on mi pomógł na objawy depresyjne, mogłem dzięki niemu wstac z łóżka i byłem ciekawy życia, dodał mi energii i chęci, które utrzymały się nawet po odstawieniu. Niestety na PSSD nie pomógl, a to chyba dlatego, że wtedy nie wiedziałem, że pomagają tylko dawki rzędu 300mg i więcej, a ja bralem 150mg. Mialem jeszcze krótkie przygody z innymi specyfikami ale to szybko się kończylo ze względu na skutki uboczne. I tak problem trwa nadal... -- 31 paź 2012, 10:09 -- Za QueenForever: - ''Zmiany obejmują zmniejszoną podwzgórze-przysadka-jąder osi ( HPTA ) funkcji [ 27 ] zmniejszyły testosteronu poziomy, [ 28 ] zmniejszenie liczby plemników, które wykazały znaczną poprawę po odstawieniu [ 29 ] i zmniejszona jakość nasienia z uszkodzonym DNA plemników, co jest odwracalne po odstawienie. [ 30 ] [ edytuj ] Skutki długoterminowe'' tu bym proponował coś na zwiększenie produkcji testosteronu, zwlaszcza wolnego są do tego celu dobre boostery tescia dla kulturystów, calkiem naturalne i dzialają!!! - ''Leczenie fluoksetyną ( Prozac ) wykazano powodują trwałe odczulanie z 5HT1a receptorów po usunięciu. SSRI szczurów [ 31 ] Długoterminowe zmiany adaptacyjne w receptorów 5-HT'' - ''Jednym z możliwych mechanizmów jest hamowanie dopaminergicznej neurotransmisji, [ 46 ], w wyniku opisanej przetrwałym dysfunkcji seksualnej.'' tu myślę ze moglby coś pomóc BUSPIRON który jest agonistą receptora 5-HT1a, a także antagonistą receptorów dopaminoergicznych D2,